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Folding@Home Information

Stanford's goal: to understand protein folding, protein aggregation, and related diseases.



What are proteins and why do they "fold"? Proteins are biology's workhorses -- its "nanomachines." Before proteins can carry out their biochemical function, they remarkably assemble themselves, or "fold." The process of protein folding, while critical and fundamental to virtually all of biology, remains a mystery. Moreover, perhaps not surprisingly, when proteins do not fold correctly (i.e. "misfold"), there can be serious effects, including many well known diseases, such as Alzheimer's, Mad Cow (BSE), CJD, ALS, and Parkinson's disease.

What does Folding@Home do? Folding@Home is a distributed computing project which studies protein folding, misfolding, aggregation, and related diseases. Stanford uses novel computational methods and large scale distributed computing, to simulate timescales thousands to millions of times longer than previously achieved. This has allowed us to simulate folding for the first time, and to now direct Stanford's approach to examine folding related disease.



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[H]ard|Folding News Articles
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  • News Articles: 142
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From Vijay over in the Stanford Forums.
....
CIWS


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Posted: Fri Apr 23, 2004 12:00 am
From Vijay over in the Stanford Forums.

"As announced in the previous thread, the new points are in. You can see the details of how we determine points in the FAQ."

http://www.stanford.edu/group/pandegroup/folding/faq.html#stats.howcred
Genetically modified bugs could fight ca....
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Posted: Fri Apr 23, 2004 12:00 am
Genetically modified bugs could fight cancer

Genetically engineered bacteria could become the latest weapons against cancer, Cancer Research UK scientists suggest in a study published in Gene Therapy Advance Online Publication.

The authors found that infecting tumours with modified forms of E. coli – famous for causing food poisoning but normally harmless – could be a safe and highly effective way of attacking cancer cells.

http://www.medicalnewstoday.com/index.php?newsid=7567
Inactivation of Alzheimer's disease gen....
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Posted: Fri Apr 02, 2004 12:00 am
Inactivation of Alzheimer's disease genes in mice causes dementia and brain degeneration.

Using advanced genetic engineering technology, scientists have discovered that deletion of two related genes linked to inherited forms of Alzheimer's disease causes memory loss and gradual death of nerve cells in the mouse brain. The researchers identified the molecular pathways through which inactivation of these genes in mice produces abnormalities strikingly reminiscent of Alzheimer's disease. The study, reported online in Neuron, may provide a new way to look at the cause of Alzheimer's disease, as it shows that the protein products of these Alzheimer's disease genes are essential for normal learning, memory and nerve cell survival.

http://www.medicalnewstoday.com/index.php?newsid=6951
Scientists Compare Rat Genome With Human....
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Posted: Fri Apr 02, 2004 12:00 am
Scientists Compare Rat Genome With Human and Mouse.

Analysis Yields New Insights into Medical Model, Evolutionary Process

Bethesda, Maryland (USA) — An international research team, supported by the National Institutes of Health (NIH), today announced it has completed a high-quality, draft sequence of the genome of the laboratory rat, and has used that data to explore how the rat's genetic blueprint stacks up against those of mice and humans.

http://www.medicalnewstoday.com/index.php?newsid=6969
A New Hypothesis About Alzheimer's Di....
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Posted: Wed Mar 17, 2004 12:00 am
A New Hypothesis About Alzheimer's Disease

A group of scientists at The Scripps Research Institute has proposed a new theory about the cause of Alzheimer's disease, the progressive neurodegenerative disorder that currently afflicts some 4.5 million Americans.

According to the hypothesis, the disease arises as a consequence of inflammation, which creates abnormal metabolites out of normal brain molecules.

These abnormal metabolites then modify "amyloid beta" proteins in the brain and cause them to misfold. Misfolded amyloid beta proteins are thought to be a major player in Alzheimer's disease, because they can accumulate into the fibrils and plaques that autopsies reveal in the brains of patients with the disease. These fibrils and plaques and their precursors are implicated in neuronal loss.

http://www.sciencedaily.com/releases/2004/03/040316072210.htm
  • Stickies: 0
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